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Inflammation increases the risk of pancreatic most cancers onset, even a quantity of years after full restoration. Combining single nucleus RNA/ATAC sequencing, mouse modeling and epigenomic profiling we are interrogating the metabolic determinants of pancreatic most cancers susceptibility after recovery from acute inflammation. The conversion is mediated by ACLY, which has been proven to be essential for cell differentiation and stemness, both in physiological and pathological conditions. However, what determines efflux of citrate from mitochondria just isn’t clear. Using histone acetylation and cell de-differentiation as read-outs, we’re interrogating mobile metabolites that might regulate the destiny of mitochondrial …